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Study · Measured the body's own peptide · 2025

2 results

The Prognostic Significance of TMSB4X in Glioma Patients.

Li S, Fan T, Hao Z, Zhang Z, Han X, Li J, Wang Y, Zhou H, Xue X

International journal of general medicine6 Jun 2025Cited by 2 (Europe PMC)

PMID40502330 (opens in a new tab)PMCIDPMC12152311 (opens in a new tab)DOI10.2147/ijgm.s521390 (opens in a new tab)PepBankPUB-40502330
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Summary

The text of this summary is not shown here, because the article license does not allow reuse. Read it at the source (opens in a new tab)

Text rights: CC BY-NC. Results still quote short attributed sentences.

Results from this study2

No result from this study comes from a test of the peptide.

Natural levels measured2

These results measure the body's own peptide. Nobody was given the peptide. They are not evidence about taking it. They are left out of key results, evidence counts and charts.

  1. 01HumanEndogenous levelObservational studyOther

    In patients with glioma, TMSB4X gene expression was an independent prognostic indicator.

    In plain wordsIn people with glioma, the activity of the gene for the body's own thymosin beta‑4 predicted outcome independently of other factors.

    Peptide
    Thymosin beta-4 (human)
    Outcome
    Direction not reportedIndependent prognostic value of TMSB4X in glioma
    Study

    n: not reported in source

    Outcome group
    Cancer biomarkers and prognosis · Cancer

    Source sentences

    Summary
    Independent prognostic analyses show that the TMSB4X gene was identified as an independent prognostic indicator for gliomas.
    1 context sentence
    Summary · context
    We explored the expression of TMSB4X by analyzing datasets from The Cancer Genome Atlas (TCGA) datasets and the Chinese Glioma Genome Atlas (CGGA).

    This result measures the body's own Thymosin beta-4 (human). Nobody was given the peptide. It is not evidence about taking it.

  2. 02Computer modelEndogenous levelIn silicoMechanism

    In GO/KEGG and GSEA analyses of glioma datasets, TMSB4X may up-regulate I-kappaB kinase/NF-kappaB signaling.

    In plain wordsComputer analysis of glioma data suggests the gene for the body's own thymosin beta‑4 may raise NF-kappaB signaling.

    Peptide
    Thymosin beta-4 (human)
    Outcome
    IncreaseTMSB4X link to I-kappaB kinase/NF-kappaB signaling
    Study

    humann: not reported in source

    Outcome group
    Receptor activity and mechanism · Cancer

    Source sentences

    Summary
    In GOKEGG and GSEA analyses, the analyses indicate that TMSB4X may play a crucial role in glioma patients by up-regulating I-kappaB kinase/NF-kappaB signaling.
    1 context sentence
    Summary · context
    TCGA datasets was utilized for Gene Set Enrichment Analysis (GSEA) to obtain potential NF-κB-related pathways in gliomas.

    This result measures the body's own Thymosin beta-4 (human). Nobody was given the peptide. It is not evidence about taking it.

AI wrote and checked these results. AI can make mistakes, so check important results against the source.

Version history

PepBank keeps every version of the data on this study. Status changes from Retraction Watch, Europe PMC and Crossref are listed with their source.

  1. Version 2Current

    13 Oct 2026, 01:52 UTC

    Study results updated

  2. Version 1

    12 Oct 2026, 18:17 UTC

    First added · source update from Europe PMC

Subjects

Terms as the source lists them.

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