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Study · Measured the body's own peptide · 2021

3 results

Expression Profile of Mouse Gm20594, Nuclear-Encoded Humanin-Like Gene.

Kim J, Choi JW, Namkung J

Journal of lifestyle medicine1 Jan 2021Cited by 6 (Europe PMC)

Summary

The text of this summary is not shown here, because the article license does not allow reuse. Read it at the source (opens in a new tab)

Text rights: CC BY-NC. Results still quote short attributed sentences.

Results from this study3

No result from this study comes from a test of the peptide.

Natural levels measured3

These results measure the body's own peptide. Nobody was given the peptide. They are not evidence about taking it. They are left out of key results, evidence counts and charts.

  1. 01AnimalEndogenous levelAnimal study (in vivo)Other

    In C57BL6/J mice, the humanin-like gene Gm20594 was highly expressed in brown adipose tissue.

    In plain wordsIn mice, the gene for the body's own humanin-like peptide was highly active in brown fat tissue.

    Peptide
    Humanin
    Outcome
    Not applicableGm20594 expression in brown adipose tissue
    Study

    mousen: not reported in source

    C57BL6/J mouse tissues
    Outcome group
    Mitochondrial function · Whole body

    Source sentences

    Summary
    Both Mtrnr1 and Gm20594 were highly expressed in brown adipose tissue.

    This result measures the body's own Humanin. Nobody was given the peptide. It is not evidence about taking it.

  2. 02LabEndogenous levelIn vitro (cells)Other

    In differentiating C2C12 myocytes, a model of mitochondrial biogenesis, Gm20594 was upregulated.

    In plain wordsIn muscle cells growing in the lab, the gene for the body's own humanin-like peptide became more active as the cells matured.

    Peptide
    Humanin
    Outcome
    IncreaseGm20594 expression during myocyte differentiation
    Study

    mousen: not reported in source

    C2C12 myocytes
    Outcome group
    Mitochondrial function · Whole body

    Source sentences

    Summary
    In models of mitochondrial biogenesis, both Mtrnr1 and Gm20594 were upregulated during myocyte and brown adipocyte differentiation.

    This result measures the body's own Humanin. Nobody was given the peptide. It is not evidence about taking it.

  3. 03LabEndogenous levelIn vitro (cells)Other

    In differentiating brown adipocytes, a model of mitochondrial biogenesis, Gm20594 was upregulated.

    In plain wordsIn brown fat cells growing in the lab, the gene for the body's own humanin-like peptide became more active as the cells matured.

    Peptide
    Humanin
    Outcome
    IncreaseGm20594 expression during brown adipocyte differentiation
    Study

    mousen: not reported in source

    Brown adipocytes
    Outcome group
    Mitochondrial function · Whole body

    Source sentences

    Summary
    In models of mitochondrial biogenesis, both Mtrnr1 and Gm20594 were upregulated during myocyte and brown adipocyte differentiation.

    This result measures the body's own Humanin. Nobody was given the peptide. It is not evidence about taking it.

AI wrote and checked these results. AI can make mistakes, so check important results against the source.

Version history

PepBank keeps every version of the data on this study. Status changes from Retraction Watch, Europe PMC and Crossref are listed with their source.

  1. Versions 4 to 18Current

    28 Oct 2026, 02:02 UTC

    The same notice was added 15 times, first on 13 Oct 2026, 21:44 UTC. The status did not change between them.

    Study results updated

  2. Version 3

    13 Oct 2026, 16:31 UTC

    Study data updated · source update from Europe PMC

  3. Version 2

    10 Oct 2026, 08:19 UTC

    Study results updated

  4. Version 1

    9 Oct 2026, 02:47 UTC

    First added · source update from Europe PMC

Subjects

Terms as the source lists them.

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