In a CD-1 db/db mouse model of diabetic kidney fibrosis, thymosin β4 levels were lower in CD-1 mice.
In plain wordsIn mice, the body's own thymosin β4 levels were lower in a diabetic CD-1 mouse strain.
- Peptide
- Thymosin beta-4 (human)
- Outcome
- DecreaseTβ4 levels in CD-1 mice
- Study
mousen: not reported in source
CD-1 db/db mice- Outcome group
- Inflammation markers · Metabolic and weight
Source sentences
Thymosin β4 levels were also lower in CD-1 mice.
1 context sentence(design, population, supporting numbers)
The gene expression of prolyl oligopeptidase, the enzyme essential for antifibrotic peptide N-acetyl-seryl-aspartyl-lysyl-proline production from thymosin β4, was significantly lower in the CD-1 mice.
This result measures the body's own Thymosin beta-4 (human). Nobody was given the peptide. It is not evidence about taking it.