In an LPS-induced model of acute lung injury, the lung's own thymosin β4 level fell after LPS administration.
In plain wordsIn an animal model of lung injury, the body's own thymosin beta‑4 in the lung fell after LPS.
- Peptide
- Thymosin beta-4 (human)
- Outcome
- DecreasePulmonary thymosin beta-4 level after LPS
- Study
n: not reported in source
LPS-induced acute lung injury model- Outcome group
- Inflammation markers · Respiratory
Source sentences
Pulmonary levels of E-cadherin, vascular endothelial-cadherin, occludin, myosin phosphatase target subunit 1, and thymosin β4 were decreased
1 context sentence(design, population, supporting numbers)
We investigated the dynamic alterations and roles of actomyosin in ALI in vivo and in vitro models induced by LPS.
This result measures the body's own Thymosin beta-4 (human). Nobody was given the peptide. It is not evidence about taking it.