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Study · Ex vivo tissue · 2016

1 result

Defective postreperfusion metabolic recovery directly associates with incident delayed graft function.

Wijermars LG, Schaapherder AF, de Vries DK, Verschuren L, Wüst RC, Kostidis S, Mayboroda OA, Prins F, Ringers J, Bierau J, Bakker JA, Kooistra T, Lindeman JH

Kidney international14 May 2016Cited by 34 (Europe PMC)

Summary

The text of this summary is not shown here, because the article license does not allow reuse. Read it at the source (opens in a new tab)

Text rights: Publisher copyright. Results still quote short attributed sentences.

Results from this study1

1 result from studies that gave the peptide. No result from this study comes from people.

  1. 01LabEx vivo tissueEfficacy outcome

    In human kidney biopsies, SS‑31 significantly preserved mitochondrial function after simulated ischemia reperfusion.

    In plain wordsIn lab tests on human kidney tissue, SS‑31 helped keep mitochondria working after simulated blood-flow interruption and return.

    Outcome
    IncreaseMitochondrial function after simulated ischemia reperfusion
    Study

    humann: not reported in source

    Human kidney biopsies with simulated ischemia reperfusion
    Outcome group
    Mitochondrial function · Kidney and urinary
    Studied dose
    Dose not stated

    Source sentences

    Summary
    The archetypical mitochondrial stabilizing peptide SS-31 significantly preserved mitochondrial function in human kidney biopsies following simulated ischemia reperfusion.

AI wrote and checked these results. AI can make mistakes, so check important results against the source.

Version history

PepBank keeps every version of the data on this study. Status changes from Retraction Watch, Europe PMC and Crossref are listed with their source.

  1. Version 2Current

    10 Oct 2026, 08:19 UTC

    Study results updated

  2. Version 1

    9 Oct 2026, 02:47 UTC

    First added · source update from Europe PMC

Subjects

Terms as the source lists them.

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