Study · In vitro (cells) · 2013
6 resultsJeon BJ, Yang Y, Kyung Shim S, Yang HM, Cho D, Ik Bang S
Experimental cell research25 May 2013Cited by 24 (Europe PMC)
The text of this summary is not shown here, because the article license does not allow reuse. Read it at the source (opens in a new tab)
Text rights: Publisher copyright. Results still quote short attributed sentences.
6 results from studies that gave the peptide. No result from this study comes from people.
In human adipose-derived stem cells, exogenous Tβ4 enhanced proliferation.
In plain wordsIn cells, Tβ4 increased the growth of human fat-derived stem cells.
humann: not reported in source
Adipose tissue-derived stem cells (ASCs)Source sentences
Exogenous Tβ4 treatment enhanced the proliferation of human ASCs
In human adipose-derived stem cells, exogenous Tβ4 produced prominent nuclear localization of PCNA immunoreactivity.
In plain wordsIn cells, Tβ4 raised nuclear PCNA, a marker of cell division, in human fat-derived stem cells.
humann: not reported in source
Adipose tissue-derived stem cells (ASCs)Source sentences
Exogenous Tβ4 treatment enhanced the proliferation of human ASCs, resulting in prominent nuclear localization of PCNA immunoreactivity.
In human adipose-derived stem cells, exogenous Tβ4 increased IL-8 secretion.
In plain wordsIn cells, Tβ4 increased IL-8 release from human fat-derived stem cells.
humann: not reported in source
Adipose tissue-derived stem cells (ASCs)Source sentences
exogenous Tβ4 also increased IL-8 secretion and blocking of IL-8 with neutralizing antibodies decreased Tβ4-induced ASC proliferation, suggesting that IL-8 is a critical mediator of Tβ4-enhanced proliferation.
In human adipose-derived stem cells, neutralizing antibodies against IL-8 decreased the Tβ4-induced proliferation.
In plain wordsIn cells, blocking IL-8 reduced the extra growth that Tβ4 caused in human fat-derived stem cells.
humann: not reported in source
Adipose tissue-derived stem cells (ASCs)Source sentences
blocking of IL-8 with neutralizing antibodies decreased Tβ4-induced ASC proliferation
In human adipose-derived stem cells, Tβ4 activated phosphorylation of ERK1/2.
In plain wordsIn cells, Tβ4 switched on the ERK1/2 signalling step in human fat-derived stem cells.
humann: not reported in source
Adipose tissue-derived stem cells (ASCs)Source sentences
Moreover, Tβ4 activated phosphorylation of ERK1/2 and increased the nuclear translocation of NF-κB.
In human adipose-derived stem cells, Tβ4 increased the nuclear translocation of NF-κB.
In plain wordsIn cells, Tβ4 increased movement of NF-κB into the nucleus in human fat-derived stem cells.
humann: not reported in source
Adipose tissue-derived stem cells (ASCs)Source sentences
Moreover, Tβ4 activated phosphorylation of ERK1/2 and increased the nuclear translocation of NF-κB.
AI wrote and checked these results. AI can make mistakes, so check important results against the source.
PepBank keeps every version of the data on this study. Status changes from Retraction Watch, Europe PMC and Crossref are listed with their source.
Version 2Current
13 Oct 2026, 01:52 UTC
Study results updated
Version 1
12 Oct 2026, 18:17 UTC
First added · source update from Europe PMC
Terms as the source lists them.
PepBank is a research reference. Talk to a doctor before you use any peptide.
In PepBank now:147 peptides, 6 blends and 13 other compounds97,577 study results from 17,383 studies8,473 clinical trialsUpdated