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Study · Measured the body's own peptide · 2007

1 result

Investigations into the influence and role of tissue factor in the pathogenesis of myocardial hypertrophy

Frentzou GA

University of Hull1 Jan 2007

PepBankPUB-93721010
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Summary

The text of this summary is not shown here, because the article license does not allow reuse. Read it at the source (opens in a new tab)

Text rights: Publisher copyright. Results still quote short attributed sentences.

Results from this study1

No result from this study comes from a test of the peptide.

Natural levels measured1

These results measure the body's own peptide. Nobody was given the peptide. They are not evidence about taking it. They are left out of key results, evidence counts and charts.

  1. 01LabEndogenous levelIn vitro (cells)Mechanism

    In H9c2 cardiomyocytes, stimulation with exogenous tissue factor up-regulated expression of mechano growth factor.

    In plain wordsIn cells, tissue factor raised the body's own mechano growth factor levels in heart muscle cells.

    Peptide
    MGF / PEG-MGF
    Outcome
    IncreaseMechano growth factor expression after tissue factor stimulation
    Study

    n: not reported in source

    H9c2 cardiomyocytes
    Comparator
    Unstimulated cells
    Outcome group
    Receptor activity and mechanism · Cardiovascular

    Source sentences

    Summary
    Stimulation of H9c2 cardiomyocytes with exogenous TF resulted in the up-regulation of mechano growth factor.

    This result measures the body's own MGF / PEG-MGF. Nobody was given the peptide. It is not evidence about taking it.

AI wrote and checked these results. AI can make mistakes, so check important results against the source.

Version history

PepBank keeps every version of the data on this study. Status changes from Retraction Watch, Europe PMC and Crossref are listed with their source.

  1. Version 2Current

    13 Oct 2026, 21:44 UTC

    Study results updated

  2. Version 1

    13 Oct 2026, 16:31 UTC

    First added · source update from Europe PMC

Subjects

Terms as the source lists them.

Publication types

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