In atRAL-exposed mouse photoreceptor cells, colivelin may significantly reduce ferroptosis through ACSL4 and PTGS2 regulation.
In plain wordsIn cells exposed to atRAL, colivelin may reduce ferroptosis.
- Peptide
- Colivelin
- Outcome
- DecreaseFerroptosis
- Study
mousen: not reported in source
atRAL-exposed 661W photoreceptor cells- Outcome group
- Neuroprotection · Eye
Source sentences
Additionally, our findings further confirm that STAT3 activator Colivelin may significantly reduce ferroptosis in 661W photoreceptor cells exposed to atRAL by regulating the ACSL4 and PTGS2 gene.
1 context sentence(design, population, supporting numbers)
In this study, we explore the involvement of signal transducer and activator of transcription 3 (STAT3) in the regulation of atRAL-induced 661W photoreceptor cells (mouse-derived photoreceptor cells) ferroptosis.
In this study, Colivelin was a lab tool or a carrier, part of a mix, or the subject of a lab method. This result is not evidence about taking Colivelin.