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Study · Not a test of the peptide alone · 2023

3 results

Analysis of the improvement effect of Astragalus extract on oxidative stress injury in viral myocarditis through STAT3/IL-6.

Chang Z, Zhang Y, Wang L, Wang H, Tian L, Qiao J

Cellular and molecular biology (Noisy-le-Grand, France)31 Dec 2023

PMID38279453 (opens in a new tab)DOI10.14715/cmb/2023.69.15.31 (opens in a new tab)PepBankPUB-38279453
Publisher pagePubMedAsk about this study
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Summary

The text of this summary is not shown here, because the article license does not allow reuse. Read it at the source (opens in a new tab)

Text rights: CC BY-NC-ND. Results still quote short attributed sentences.

Results from this study3

No result from this study comes from a test of the peptide.

Studies that did not test the peptide alone3

In this study, Colivelin was a lab tool or a carrier, part of a mix, or the subject of a lab method. These results are not evidence about taking Colivelin.

  1. 01LabNot a test of Colivelin aloneIn vitro (cells)Mechanism

    In myocarditis model cells, colivelin significantly aggravated apoptosis.

    In plain wordsIn cells in a myocarditis model, colivelin increased programmed cell death.

    Peptide
    Colivelin
    Outcome
    IncreaseApoptosis
    Study

    n: not reported in source

    CVB3-exposed HL-1 cardiomyocytes
    Outcome group
    Cell viability and death · Cardiovascular

    Source sentences

    Summary
    Colivelin, a specific activator of STAT3, further aggravated the apoptosis, inflammatory reaction and OS response of MYO cells (P < 0.05)
    1 context sentence
    Summary · context
    For this purpose, The MYO model cells prepared by intervening cardiomyocyte HL-1 with Coxsackievirus B3 (CVB3)

    In this study, Colivelin was a lab tool or a carrier, part of a mix, or the subject of a lab method. This result is not evidence about taking Colivelin.

  2. 02LabNot a test of Colivelin aloneIn vitro (cells)Mechanism

    In myocarditis model cells, colivelin significantly aggravated the inflammatory reaction.

    In plain wordsIn cells in a myocarditis model, colivelin increased the inflammatory reaction.

    Peptide
    Colivelin
    Outcome
    IncreaseInflammatory reaction
    Study

    n: not reported in source

    CVB3-exposed HL-1 cardiomyocytes
    Outcome group
    Inflammation markers · Immune and inflammation

    Source sentences

    Summary
    Colivelin, a specific activator of STAT3, further aggravated the apoptosis, inflammatory reaction and OS response of MYO cells (P < 0.05)
    1 context sentence
    Summary · context
    For this purpose, The MYO model cells prepared by intervening cardiomyocyte HL-1 with Coxsackievirus B3 (CVB3)

    In this study, Colivelin was a lab tool or a carrier, part of a mix, or the subject of a lab method. This result is not evidence about taking Colivelin.

  3. 03LabNot a test of Colivelin aloneIn vitro (cells)Mechanism

    In myocarditis model cells, colivelin significantly aggravated the oxidative stress response.

    In plain wordsIn cells in a myocarditis model, colivelin increased the oxidative stress response.

    Peptide
    Colivelin
    Outcome
    IncreaseOxidative stress response
    Study

    n: not reported in source

    CVB3-exposed HL-1 cardiomyocytes
    Outcome group
    Oxidative stress · Cardiovascular

    Source sentences

    Summary
    Colivelin, a specific activator of STAT3, further aggravated the apoptosis, inflammatory reaction and OS response of MYO cells (P < 0.05)
    1 context sentence
    Summary · context
    For this purpose, The MYO model cells prepared by intervening cardiomyocyte HL-1 with Coxsackievirus B3 (CVB3)

    In this study, Colivelin was a lab tool or a carrier, part of a mix, or the subject of a lab method. This result is not evidence about taking Colivelin.

AI wrote and checked these results. AI can make mistakes, so check important results against the source.

Version history

PepBank keeps every version of the data on this study. Status changes from Retraction Watch, Europe PMC and Crossref are listed with their source.

  1. Version 2Current

    27 Oct 2026, 18:20 UTC

    Study results updated

  2. Version 1

    27 Oct 2026, 16:32 UTC

    First added · source update from Europe PMC

Subjects

Terms as the source lists them.

Publication types

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