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Study · Not a test of the peptide alone · 2010

2 results

Prevention of the alcohol-induced changes in brain-derived neurotrophic factor expression using neuroprotective peptides in a model of fetal alcohol syndrome.

Incerti M, Vink J, Roberson R, Benassou I, Abebe D, Spong CY

American journal of obstetrics and gynecology1 May 2010Cited by 13 (Europe PMC)

PMID20452488 (opens in a new tab)DOI10.1016/j.ajog.2010.03.050 (opens in a new tab)PepBankPUB-20452488
Publisher pagePubMedAsk about this study
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Summary

The text of this summary is not shown here, because the article license does not allow reuse. Read it at the source (opens in a new tab)

Text rights: Publisher copyright. Results still quote short attributed sentences.

Results from this study2

No result from this study comes from a test of the peptide.

Studies that did not test the peptide alone2

In this study, Davunetide was a lab tool or a carrier, part of a mix, or the subject of a lab method. These results are not evidence about taking Davunetide.

  1. 01AnimalNot a test of Davunetide aloneAnimal study (in vivo)Mechanism

    In mouse embryos, NAP plus SAL prevented the alcohol-induced BDNF expression increase at 24 hours.

    In plain wordsIn mouse embryos, NAP plus SAL prevented the BDNF increase at 24 hours compared with alcohol exposure alone.

    Peptide
    Davunetide
    Outcome
    DecreaseBDNF expression at 24 hours
    Study

    mousen: not reported in source

    Prenatal alcohol-exposed mouse embryos
    Comparator
    Alcohol exposure without NAP plus SAL
    Outcome group
    Neuroprotection · Nervous system and cognition

    Source sentences

    Summary
    Pretreatment with NAP plus SAL prevented the alcohol-induced rise in BDNF expression (P<.05) at 24 hours and 10 days after alcohol exposure.
    1 context sentence
    Summary · context
    Using a well-characterized fetal alcohol syndrome (FAS) model, timed pregnant C57BL6/J mice were treated on gestational day (E) 8 with alcohol (0.03 mL/g), placebo, or alcohol plus (NAP plus SAL).

    In this study, Davunetide was a lab tool or a carrier, part of a mix, or the subject of a lab method. This result is not evidence about taking Davunetide.

  2. 02AnimalNot a test of Davunetide aloneAnimal study (in vivo)Mechanism

    In mouse embryos, NAP plus SAL prevented the alcohol-induced BDNF expression increase at 10 days.

    In plain wordsIn mouse embryos, NAP plus SAL prevented the BDNF increase at 10 days compared with alcohol exposure alone.

    Peptide
    Davunetide
    Outcome
    DecreaseBDNF expression at 10 days
    Study

    mousen: not reported in source

    Prenatal alcohol-exposed mouse embryos
    Comparator
    Alcohol exposure without NAP plus SAL
    Outcome group
    Neuroprotection · Nervous system and cognition

    Source sentences

    Summary
    Pretreatment with NAP plus SAL prevented the alcohol-induced rise in BDNF expression (P<.05) at 24 hours and 10 days after alcohol exposure.
    1 context sentence
    Summary · context
    Using a well-characterized fetal alcohol syndrome (FAS) model, timed pregnant C57BL6/J mice were treated on gestational day (E) 8 with alcohol (0.03 mL/g), placebo, or alcohol plus (NAP plus SAL).

    In this study, Davunetide was a lab tool or a carrier, part of a mix, or the subject of a lab method. This result is not evidence about taking Davunetide.

AI wrote and checked these results. AI can make mistakes, so check important results against the source.

Version history

PepBank keeps every version of the data on this study. Status changes from Retraction Watch, Europe PMC and Crossref are listed with their source.

  1. Version 2Current

    27 Oct 2026, 18:20 UTC

    Study results updated

  2. Version 1

    27 Oct 2026, 16:32 UTC

    First added · source update from Europe PMC

Subjects

Terms as the source lists them.

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