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Study · Measured the body's own peptide · 2006

2 results

Muscle IGF-I Ea, MGF, and myostatin mRNA expressions after compensatory overload in hypophysectomized rats.

Yamaguchi A, Fujikawa T, Shimada S, Kanbayashi I, Tateoka M, Soya H, Takeda H, Morita I, Matsubara K, Hirai T

Pflugers Archiv : European journal of physiology29 Aug 2006Cited by 21 (Europe PMC)

PMID16941139 (opens in a new tab)DOI10.1007/s00424-006-0127-9 (opens in a new tab)PepBankPUB-16941139
Publisher pagePubMedAsk about this study
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Summary

The text of this summary is not shown here, because the article license does not allow reuse. Read it at the source (opens in a new tab)

Text rights: Publisher copyright. Results still quote short attributed sentences.

Results from this study2

No result from this study comes from a test of the peptide.

Natural levels measured2

These results measure the body's own peptide. Nobody was given the peptide. They are not evidence about taking it. They are left out of key results, evidence counts and charts.

  1. 01AnimalEndogenous levelAnimal study (in vivo)Mechanism

    In normal rats, plantaris MGF mRNA 3 days after overload was significantly higher than in normal controls.

    In plain wordsIn rats, the body's own muscle MGF messenger RNA was higher 3 days after overload than in controls.

    Peptide
    MGF / PEG-MGF
    Outcome
    IncreaseMuscle MGF mRNA after overload, normal rats
    Study

    ratn: not reported in source

    plantaris muscle after compensatory overload
    Comparator
    Normal-control
    Outcome group
    Muscle injury and repair · Musculoskeletal

    Source sentences

    Summary
    IGF-I Ea and MGF mRNAs in normal-overloaded and hypophysectomized-overloaded 3 days after overload were significantly higher than those of normal-control and hypophysectomized-control, respectively.
    2 context sentences
    Summary · context
    To determine whether IGF-I Ea, MGF, and myostatin mRNAs are related to GH-independent overload-induced muscle growth, we examined the expressions of IGF-I Ea and MGF mRNAs in the plantaris muscle after compensatory overload in hypophysectomized rats.
    Summary · context
    The muscles were divided into four groups: normal-control, normal-overloaded, hypophysectomized-control, and hypophysectomized-overloaded.

    This result measures the body's own MGF / PEG-MGF. Nobody was given the peptide. It is not evidence about taking it.

  2. 02AnimalEndogenous levelAnimal study (in vivo)Mechanism

    In hypophysectomized rats, plantaris MGF mRNA 3 days after overload was significantly higher than in hypophysectomized controls.

    In plain wordsIn rats without a pituitary gland, the body's own muscle MGF messenger RNA was higher 3 days after overload than in controls.

    Peptide
    MGF / PEG-MGF
    Outcome
    IncreaseMuscle MGF mRNA after overload, hypophysectomized rats
    Study

    ratn: not reported in source

    plantaris muscle after compensatory overload
    Comparator
    Hypophysectomized-control
    Outcome group
    Muscle injury and repair · Musculoskeletal

    Source sentences

    Summary
    IGF-I Ea and MGF mRNAs in normal-overloaded and hypophysectomized-overloaded 3 days after overload were significantly higher than those of normal-control and hypophysectomized-control, respectively.
    2 context sentences
    Summary · context
    To determine whether IGF-I Ea, MGF, and myostatin mRNAs are related to GH-independent overload-induced muscle growth, we examined the expressions of IGF-I Ea and MGF mRNAs in the plantaris muscle after compensatory overload in hypophysectomized rats.
    Summary · context
    The muscles were divided into four groups: normal-control, normal-overloaded, hypophysectomized-control, and hypophysectomized-overloaded.

    This result measures the body's own MGF / PEG-MGF. Nobody was given the peptide. It is not evidence about taking it.

AI wrote and checked these results. AI can make mistakes, so check important results against the source.

Version history

PepBank keeps every version of the data on this study. Status changes from Retraction Watch, Europe PMC and Crossref are listed with their source.

  1. Version 2Current

    13 Oct 2026, 21:44 UTC

    Study results updated

  2. Version 1

    13 Oct 2026, 16:31 UTC

    First added · source update from Europe PMC

Subjects

Terms as the source lists them.

Publication types

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