Skip to content

Study · Measured the body's own peptide · 2005

2 results

Neuronal nitric oxide synthase inhibition differentially affects oxytocin and vasopressin secretion in salt loaded rats.

Ventura RR, Giusti-Paiva A, Gomes DA, Elias LL, Antunes-Rodrigues J

Neuroscience letters18 Jan 2005Cited by 27 (Europe PMC)

PMID15823420 (opens in a new tab)DOI10.1016/j.neulet.2004.12.032 (opens in a new tab)PepBankPUB-15823420
Publisher pagePubMedAsk about this study
Jump to 2 results

Summary

The text of this summary is not shown here, because the article license does not allow reuse. Read it at the source (opens in a new tab)

Text rights: Publisher copyright. Results still quote short attributed sentences.

Results from this study2

No result from this study comes from a test of the peptide.

Natural levels measured2

These results measure the body's own peptide. Nobody was given the peptide. They are not evidence about taking it. They are left out of key results, evidence counts and charts.

  1. 01AnimalEndogenous levelAnimal study (in vivo)Other

    In rats, short and long-term salt loading increased plasma oxytocin levels.

    In plain wordsIn rats, salt loading raised the body's own oxytocin levels in blood.

    Peptide
    Oxytocin
    Outcome
    IncreasePlasma oxytocin with salt loading
    Study

    ratn: not reported in source

    Short and long-term salt loading
    Outcome group
    Endogenous peptide levels · Endocrine and hormones

    Source sentences

    Summary
    Our data showed an increase of plasma OT and AVP levels in both short and long-term salt loading.
    1 context sentence
    Summary · context
    Thus, the aim of this study was to evaluate the effect of 7-nitroindazole (7-NI), a selective inhibitor of neuronal NOS, in the plasma OT and AVP levels in rats submitted to a short and long-term salt loading.

    This result measures the body's own Oxytocin. Nobody was given the peptide. It is not evidence about taking it.

  2. 02AnimalEndogenous levelAnimal study (in vivo)Other

    In salt-loaded rats, the neuronal NOS inhibitor 7-NI potentiated the salt-induced increase of plasma oxytocin.

    In plain wordsIn salt-loaded rats, the drug 7-NI made the rise in the body's own oxytocin levels larger.

    Peptide
    Oxytocin
    Outcome
    IncreasePlasma oxytocin with 7-nitroindazole during salt loading
    Study

    ratn: not reported in source

    Short and long-term salt loading
    Comparator
    Salt loading without 7-NI
    Outcome group
    Endogenous peptide levels · Endocrine and hormones

    Source sentences

    Summary
    The injection of 7-NI potentiated the increase of plasma OT induced by salt loading, but inhibited the increase of plasma AVP in the same experimental conditions.
    1 context sentence
    Summary · context
    Thus, the aim of this study was to evaluate the effect of 7-nitroindazole (7-NI), a selective inhibitor of neuronal NOS, in the plasma OT and AVP levels in rats submitted to a short and long-term salt loading.

    This result measures the body's own Oxytocin. Nobody was given the peptide. It is not evidence about taking it.

AI wrote and checked these results. AI can make mistakes, so check important results against the source.

Version history

PepBank keeps every version of the data on this study. Status changes from Retraction Watch, Europe PMC and Crossref are listed with their source.

  1. Version 2Current

    11 Oct 2026, 20:03 UTC

    Study results updated

  2. Version 1

    9 Oct 2026, 02:51 UTC

    First added · source update from Europe PMC

Subjects

Terms as the source lists them.

PepBank is a research reference. Talk to a doctor before you use any peptide.